This week in research.

Summary of works published 2026-08-03 to 2026-08-10 1) Surveillance of zoonotic and production-limiting pathogens in invasive free-ranging wild pigs in Alberta, Canada. Journal: BMC Veterinary Research. Date: 2026-08-05. Cohort authors: Sylvia Checkley; Mathieu Pruvot. - Large-scale screening (2018–2024) of 331 wild pigs for 17 pathogens, revealing widespread exposure and diverse pathogen co-occurrence. - Serology and molecular findings highlight notable prevalence of PCV2 (32.9%) and PCV3 (27.2%), with other parasites detected (e.g., Eimeria spp.). - Co-detection of two or more pathogens was common (55.9%), and older pigs showed higher risk for PCV2/PCV3 infections, underscoring implications for livestock health and public health. 2) Human neuronal cells SH-SY5Y as an in vitro model to study Neospora caninum infection and innate immune modulation. Journal: The Veterinary Journal. Date: 2026-08-05. Cohort author: Eduardo R. Cobo. - Proposes SH-SY5Y neuronal cells as an in vitro model to study Neospora caninum infection. - Aims to enable controlled investigation of host–parasite interactions and innate immune responses in neurons. - Published in The Veterinary Journal with author Eduardo R. Cobo. 3) First genetic characterization of gastrointestinal nematode communities and benzimidazole resistance in cattle from Argentina. Journal: Heredity. Date: 2026-08-03. Cohort author: John S. Gilleard. - Reports the first genetic profiling of GI nematode communities in Argentinian cattle and surveys benzimidazole resistance markers. - Provides baseline insights into species composition and anthelmintic resistance risk. - Published in Heredity with author John S. Gilleard. 4) Analysis code for: Modulating neutrophil response to injury enables enhanced skin regeneration. Venue: Zenodo (CERN European Organization for Nuclear Research). Date: 2026-08-03. Cohort author: Jeff Biernaskie. - Open-source analysis workflow accompanying Labit et al. on neutrophil modulation and skin repair (QC, sample integration, cell-type annotation, neutrophil subclustering, RNA velocity/latent time, MELD perturbation likelihood, cross-species validation). - Includes cross-reference to datasets and methodological notes; data available at GEO under GSE145403; described as code-only with unsupervised or reference-based analyses. - Published in Zenodo with author Jeff Biernaskie (two code entries described in the data lines). 5) A nerve and mast cell sentinel system releases extracellular condensates to induce macrophage repair. Venue: Open Access CRIS of the University of Bern. Date: 2026-08-03. Cohort authors: Nathan C. Peters; Johnathan Canton. - Describes a neuron–mast cell sentinel circuit where sensory neurons activate mast cells to release membraneless condensates containing over 200 proteins (including MARCO) that prompt rapid macrophage repair at injury. - Lattice light sheet microscopy reveals a degranulation step and condensate-mediated recruitment/activation of macrophages with a repair-like phenotype. - Published as Open Access CRIS from the University of Bern with authors Nathan C. Peters and Johnathan Canton. By the numbers - Journal/venue tallies (including duplicates in the data): BMC Veterinary Research 1; The Veterinary Journal 1; Heredity 1; Zenodo 2 (two code entries for the same neutrophil–skin regeneration study); Open Access CRIS of the University of Bern 1. - Total works described (unique works): 5 - Total data lines in the provided data: 6 (note: two Zenodo entries correspond to the same code release)